而超微结构改变以软骨细胞破碎的比较多见，有的软骨细胞则增生，基质胶原原纤维明显减少。
Many broken cartilage cells could be observed and matrix collagenous fibril decreases obviously, but some cartilage cells proliferated.
目的：研究3周的跑台奔跑锻炼能否刺激椎间盘细胞外基质的产生和细胞增生。
To investigate whether 3-weeks treadmill running exercise can stimulate matrix production and cellular proliferation of the IVD. Summary of Background Data.
结果发现基质成纤维细胞的增生活跃程度与上皮基底细胞的细胞层数和细胞大小密切相关。
Results Active degree of proliferation of stroma fibroblasts correlated closely with the number of epithelial basal layer cells, all as well, with the size of them.
提示，临床上LMWH能通过对增生性肾炎患者系膜细胞增殖和系膜基质积聚的抑制作用而发挥效应。
The effect of LMWH on patients with proliferative glomerulonephritis may be associated with inhibiting the proliferation of mesangial cell and the accumulation of mesangial matrix.
这种增生膜的形成主要包括细胞的增生和细胞外基质的沉积。
The process contains the proliferation of cells and the accumulation of extracellular matrix components.
目的明确静脉移植后内膜增生（IH）过程中分泌细胞外基质（ECM ）的间质效应细胞。
Objective To understand the interstitial effective cells secreting extracellular matrix (ECM) in intimal hyperplasia (IH) after venous implantation.
如果同意假说是正确的，那么抑制细胞和细胞外基质增殖就可以作为用于防止增生性瘢痕形成的治疗方法。
Accepting the hypothesis to be correct, a therapy that inhibits cell and extracellular matrix proliferation can be used to prevent the hypertrophic scars formation.
结论川芎嗪能增强免疫介导再障小鼠骨髓造血细胞和基质细胞粘附分子的表达，有利于造血细胞的增生。
Conclusions Ligustrazine can increase the expression of adherent molecule of bone marrow cells in immune mediated AA mice, thus promote the growth of hematopoietic cells.
肾小球系膜：基质轻度增生。发现部分电子致密物质，但这不是典型的免疫类沉积。
Mesangium: Possible mild increase in matrix. Some electron dense material identified but this is not typical of immune type deposits.
肝纤维化是各种致病因素导致肝内结缔组织异常增生的肝内弥散性细胞外基质过度沉积的病理过程。
Hepatic fibrosis was caused by intrahepatic connective tissue dysplasia due to many pathological factors, Which was a massive diffuse ECM deposition pathological process in human body.
肝纤维化是各种致病因素导致肝内结缔组织异常增生的肝内弥散性细胞外基质过度沉积的病理过程。
Hepatic fibrosis was caused by intrahepatic connective tissue dysplasia due to many pathological factors, Which was a massive diffuse ECM deposition pathological process in human body.
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