血栓调节蛋白可完全抑制凝血酶和利斯托司汀诱导的血小板聚集。
Thrombomodulin can entirely inhibit thrombin and ristocetin-induced platelet aggregation.
血纤维蛋白和血小板一起形成血栓。血友病就是因为遗传上缺乏一种凝血胶原引起的。
Fibrin and platelets combine to form a clot. Hemophilia is caused by a hereditary lack of one of the clotting factors.
目的观察海洋假单胞菌碱性蛋白酶（MPAP）的溶栓和抑制血栓形成的作用，研究其对纤溶、凝血和血小板聚集功能的影响。
ObjectiverTo observe the thrombolysis and anti-thrombosis effects of MPAP; To study the actions of MPAP on blood coagulation, platelet aggregation and fibrinolytic activity.
动脉血栓通常形成于血液湍流区和粥样癍块破裂区，这些部位的内皮下组织暴露于血小板和凝血蛋白。
Arterial thrombi usually form in regions of disturbed flow and at sites of rupture of an atherosclerotic plaque, which exposes the thrombogenic subendothelium to platelets and coagulation proteins;
结果：薤白不同溶剂提取物能显著延长小鼠凝血时间，提高胶原蛋白-肾上腺素血栓模型小鼠的恢复率。
Results: the extractions of different dissolvents can prolong the clotting time and increase the instauration rate of collagen-adrenaline model of mice.
结果：薤白不同溶剂提取物能显著延长小鼠凝血时间，提高胶原蛋白-肾上腺素血栓模型小鼠的恢复率。
Results: the extractions of different dissolvents can prolong the clotting time and increase the instauration rate of collagen-adrenaline model of mice.
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