结论高氧分压体外循环再氧合可导致心肌和脑组织氧自由基介导的再氧合损伤；
CONCLUSION Hyperoxic CPB leads to direct myocardial and cerebral injury induced by oxygen free radical.
肺缺血再灌注损伤可以表现为肺血管阻力升高，氧合功能下降，顺应性降低以及水肿形成等等。
The manifestations of pulmonary ischemia -reperfusion injury can be characterized by increased pulmonary vascular resistance, decreased oxygenation capacity, worsened compliance, and edema formation.
探讨血红素氧合酶1（HO 1）对大鼠肺缺血再灌注损伤（I/R）的保护作用。
To examine whether increase in Heme Oxygenase-1(HO-1) activity has protective effects on ischemia-reperfusion (I/R) injury of rat lung.
探讨血红素氧合酶1（HO 1）对大鼠肺缺血再灌注损伤（I/R）的保护作用。
To examine whether increase in Heme Oxygenase-1(HO-1) activity has protective effects on ischemia-reperfusion (I/R) injury of rat lung.
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