产生胰岛素的细胞称为β细胞。
因此人体不能够产生胰岛素。
糖尿病这种疾病损害，正常人产生胰岛素的能力。
So diabetes is a disease that affects a person's ability to produce insulin.
例如，在医学上，产生胰岛素治疗糖尿病。
For example, in medicine, insulin is produced to treat diabetes.
胰腺开始产生胰岛素。
胰腺产生胰岛素的激素控制血液中的葡萄糖量。
The pancreas produces hormone insulin which controls the amount of glucose in the blood.
高胰岛素血症可诱发家兔产生胰岛素和抗性动脉粥样硬化。
Hyperinsulinemia could induce insulin resistance and led to atherosclerosis in rabbit.
两个月后，将塑料棒取出，代之以能产生胰岛素的小猪细胞。
After two months, the rod was removed and the insulin-producing piglet cells replaced it.
糖尿病一型以前被称作青少年糖尿病﹐身体不能产生胰岛素。
In Type I diabetes, previously called juvenile diabetes, the body does not produce insulin.
灵长类动物1型糖尿病的形成发生在胰岛细胞停止共同产生胰岛素。
The primates in the current study had type 1 diabetes, the form that occurs when islet cells in the pancreas stop producing insulin all together.
一旦进入细胞的药物锁，它停止他们的破坏胰腺中产生胰岛素的细胞。
Once the drug locks onto the cells, it stops them destroying the insulin-producing cells in the pancreas.
但治疗似乎是在处理时间对剩余产生胰岛素的细胞在患者左额和年龄而定。
But the treatment appears to be dependent on the amount of residual insulin-producing cells left in the patient and their age at the time of the treatment.
但是一些专家相信是人体抵抗疾病的防御机制由于一些原因破坏了产生胰岛素的细胞。
But some experts believe the body's defenses against disease for some reason destroy the cells that produce insulin.
但别的类型的细胞区分出来还是有困难的，比如可以产生胰岛素而治疗糖尿病的细胞。
Other cell types are proving more difficult, like insulin-producing islet cells to treat diabetes.
在未来，这项移植手术可以治疗那些身体不能产生胰岛素的患者提供无限量供应的组织。
In the future, the transplant operation could provide an unlimited supply of tissue to treat people whose bodies cannot produce insulin.
什么东西将超级减肥者和其他人群分别开来？在进食后他们的身体产生胰岛素的有多么快。
What separated the super losers from the rest? How quickly their bodies produced the hormone insulin after eating.
随产生胰岛素的胰腺胰岛细胞损伤，免疫系统的应答缺陷也被认为是产生1型糖尿病的原因。
The flawed response from the immune system was understood to be the reason behind the damage caused to islet cells in the pancreas where insulin is produced.
基因修改过不能合成神经酰胺的动物在吃药的情况下也和正常动物一样，不会产生胰岛素抵抗。
Animals genetically altered in a way that limited their synthesis of ceramide didn't become insulin resistant when taking the drug as normal animals did.
脂肪组织的结疤是因为我们体重的增加，我们发现这是因为瘦素剧增，以及胰腺产生胰岛素的增加。
Scarring of fat tissue is known to increase as we gain weight and we found that this is exacerbated by leptin, as well as an increase in insulin, produced by the pancreas.
有几年，治愈的说法看来已经成真了，直到大多的受体中的移植细胞停止产生胰岛素，患者只得重新开始注射。
It looked for a few years like the real deal - until most of the transplanted cells stopped producing insulin in most recipients, and the patients had to resume taking injections.
研究人员们现在正试图确定引发这种疾病的遗传和环境因素并且就是否能让细胞产生胰岛素进行试验。
Researchers are now trying to identify genetic and environmental causes of the disease and to see if they can get cells to produce insulin.
这意味着为产生胰岛素的细胞提供稳定来源的大门已经打开，这些细胞能移植到严重的糖尿病患者中。
This opens the door to the possibility of providing a steady supply of insulin-producing cells that can be transplanted into patients affected by this serious disease.
这对于糖尿病患者是好消息。近来保健领域无意发现一种能使1型糖尿病患者恢复产生胰岛素的新方法。
Here is some good news for the diabetic patients. The health world recently stumbled upon a novel means to restore insulin production in people suffering from type 1 diabetes.
其中增长最快的科学领域，是研究JDRF支持旨在保护和再生产生胰岛素的细胞在人谁处于危险或有糖尿病。
Among the fastest-growing scientific areas that JDRF supports is research aimed at protecting and regenerating insulin-producing cells in people who are at risk or have diabetes.
对于大多数病人来说，免疫系统攻击破坏了可以产生胰岛素的胰岛细胞，许多病人必须为了生存必须注射胰岛素。
In most patients, the immune system attacks and destroys the islet cells that produce insulin. Many patients must inject themselves with insulin, simply to survive.
JDRF资助的研究显示，葡萄糖传感信号的酶生产新产生胰岛素的细胞，提示了一条新路的治疗1型糖尿病的可能性。
JDRF-funded study reveals that a glucose-sensing enzyme signals the production of new insulin-producing cells, suggesting a new path of treatment possibilities for type 1 diabetes.
JDRF资助的研究显示，葡萄糖传感信号的酶生产新产生胰岛素的细胞，提示了一条新路的治疗1型糖尿病的可能性。
JDRF-funded study reveals that a glucose-sensing enzyme signals the production of new insulin-producing cells, suggesting a new path of treatment possibilities for type 1 diabetes.
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