测定外周血血清中丙二醛（MDA）的含量。
大鼠大脑皮质和海马中一氧化氮和丙二醛含量。
Contents of nitric oxide and MDA in cerebral cortex and hippocampus of rats.
此外，还测定了细胞内丙二醛(MDA)的含量。
In addition, cellular content of malondialdehyde (MDA) was detected.
丙二醛(MDA)是脂质过氧化产物的稳定代谢物。
Molondialdehyde (MDA) was the stable metabolite of lipid peroxidation products.
肾皮质制备匀浆检测丙二醛含量和超氧化物歧化酶活性。
In renal cortex homogenate, the contents of malonaldehyde and activity of superoxide dismutase were measured.
根系和叶片发生膜脂过氧化作用，丙二醛(MDA)含量增加。
Malondialdehyde (MDA) content in roots and leaves produced by membrane lipid peroxidation was enhanced.
两种处理都可以通过降低果实丙二醛（MDA）含量来延缓其衰老。
The two treatments could delay the fruit senescence as indicated by reducing the malondialdehyde(MDA) content.
测定小鼠T淋巴细胞功能，IL-2和血清丙二醛（MDA）水平。
The spleen T lymphocyte proliferation, IL-2 production and serum MDA were measured.
测定血清丙二醛含量和总SOD活性以及肝组织匀浆中的脂褐素含量。
The content of MDA in serum and lipofuscin in liver homogenate and the activity of T SOD in serum were detected.
其作用机制可能与降低再灌注期胃组织中丙二醛的含量，减少胃酸分泌有关。
The mechanisms of protection may be related to the decrease of MDA in gastric tissue and the gastric acid output .
他在《农业和食品化学期刊》上报导说，浓缩的酒确实减少了丙二醛的形成。
As he reports in the Journal of Agricultural and Food Chemistry, the wine concentrate did indeed reduce the formation of malondialdehyde.
氧化油脂对肝胰腺中丙二醛（MDA）含量并无明显影响（P＞0.05）。
The levels of MDA were not significantly different in all fishes (P >0. 05).
测定丙二醛（MDA ）、巯基含量和总抗氧化能力等氧化还原相关生化指标。
The MDA level, thiol content, and total antioxidative capacity of cells were also measured.
并检测血糖、糖基化血清蛋白和血清脂过氧化产物丙二醛浓度以反映代谢控制。
Serum concentrations of glucose, glycosylated serum protein, and malondialdehyde, derived from lipid peroxidation were also examined to estimate metabolic control.
方法：考察醒酒时间、乙醇浓度、 丙二醛含量、谷胱甘肽过氧化物酶活力。
Methods:To investigate sleeping time, alcohol concentration, MDA content and GSH-Px activity.
背景：幼鼠脑缺氧缺血后，脑组织水肿加重，脑组织中一氧化氮及丙二醛水平增高。
BACKGROUND: After cerebral tissue ischemia and anoxia in young rats, the cerebral edema gets serious, and the levels of nitric oxide (no) and malondialdehyde (MDA) decrease.
以超氧化物岐化酶(SOD)活性和丙二醛(MDA)的含量反映脂质过氧化程度。
Superoxide Dismutase (SOD) and Malondialdehyde (MDA) were measured to show the degree of lipid peroxidation.
目的研究高压氧治疗脑梗死对血清超氧化物歧化酶和丙二醛活性的影响及其临床价值。
Objective To study the effect of hyperbaric oxygenation (HBO) therapy on the activities of serum SOD and MDA of patients with cerebral infarction and its clinical value.
探讨了丙二醛对体外培养人骨髓间充质干细胞的生长与增殖状态的影响及其作用机制。
The effects of malondialdehyde on the growth and proliferation of human bone marrow mesenchymal stem cells were studied.
主要观察指标：正常和心肌缺血家兔在急性低氧条件下血清sod和丙二醛水平比较。
MAIN OUTCOME MEASURES: Comparison of serum SOD and malondialdehyde between normal rabbits and myocardial ischemic rabbits under the acute hypothermia and hypoxia conditions.
活性氧代谢还与植物内源激素、丙二醛含量和电解质渗漏率及果实品质变化有密切关系。
The active oxygen metabolism is significantly related to the plant hormones, content of malondialdehyde (MDA), cell electrolyte leakage and quality of fruits.
按南京建成生物工程研究所提供试剂盒说明，测定脊髓匀浆丙二醛含量及超氧化物歧化酶活性。
Referring to the kit instruction offered by Nanjing Jiancheng Biological Engineering Institute, the activity of SOD and content of MDA in spinal homogenate were measured.
尤其是被称为丙二醛的这类和动脉硬化、癌症、糖尿病等一系列严重的疾病纠缠不清的一种物质。
One in particular, called malondialdehyde, is implicated in arteriosclerosis, cancer, diabetes and a host of other serious diseases.
处死小鼠后取肝、脾、肾组织及血清，测定超氧化物歧化酶（SOD）活力及丙二醛（MDA）含量。
The tissues and blood were obtained, in which the vitality of SOD and the content of MDA were detected.
目的：观察补肾益气类中药对运动大鼠血生化学指标、血清超氧化物歧化酶活性、丙二醛水平的影响。
AIM: To observe the effects of Bushen Yiqi Chinese herb on blood biochemical index, activity of serum superoxide dismutase (SOD) and level of malonaldehyde (MDA) in sport rats.
用自身对照方法，观察服灵芝前后铅、锌、铜、锰、铁、丙二醛和过氧歧化酶（SOD）活性的改变。
The effect of fonr trace elements, malonaldehyde(MDA )and superoxide dismutase (SOD) activity were observed with with. self-control method.
方法：应用高分辨率超声检测血流介导的血管扩张（FMD）及硫代巴比妥法测定丙二醛（MDA）。
Methods:Flow mediated dilation (FMD) was tested with high resolution ultrasound method and malondialdehyde (MDA) was detected by using thiobarbituric acid reactive substances (TBARS).
白茶能显著降低四氯化碳肝损伤小鼠的转氨酶和丙二醛含量，降低幅度分别为21.8%和38.5%。
White tea can separately reduce 21.8% and38.5% the contents of aminotransferase and malondialdehyde which can damage the liver.
白茶能显著降低四氯化碳肝损伤小鼠的转氨酶和丙二醛含量，降低幅度分别为21.8%和38.5%。
White tea can separately reduce 21.8% and38.5% the contents of aminotransferase and malondialdehyde which can damage the liver.
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