对照组则注射与地塞米松等体积的灭菌生理盐水。
Those in the control group underwent the same way of injection with physiological saline simultaneously.
目的比较TRH、EGF与地塞米松对未成熟胎肺形态发育及表面活性物质水平的影响。
Objective To compare the effects of TRH, EGFand Dexamethasone(Dex)on the morphogenesis and the levels of premature fetal lungs.
目的考察乳酸环丙沙星注射液与地塞米松磷酸钠注射液配伍稳定性，为临床联合用药提供依据。
AIM The compatible stability of ciprofloxacin lactate injection with dexamethasone sodium phosphate injection was strictly checked in order to provide a basis for clinical drugs.
在体外实验中，山茛菪碱对分离出的休克大鼠肝溶酶体无直接作用，提示654 - 2在整体实验中显示出的保护肝脏溶酶体作用的机理与地塞米松不同。
In vitro experiments showed that anisodamine had no direct stabilizing effect on isolated hepatic lysosomal granules from shocked rats, which effect is different from that of steroid.
地塞米松通常与枢复宁一起使用。
通过对文献的进一步分析发现，父母趋向与治疗短的地塞米松方案。
On further review of the literature, parents tend to prefer the shorter duration of therapy with Dexamethasone.
结果表明，地塞米松与长疗程的强的松有同样疗效。
The results showed that Dexamethasone was as efficacious as the longer course of Prednisone.
目的：制备醋酸地塞米松凝胶剂，考察其质量与体外释放的特征。
Objective: To prepare the dexamethasone acetate gel and investigate its quality and the character of its release in vitro.
未发现吸氧方式、表面活性物质、多巴胺和出生前地塞米松的使用、输血和母亲病情与ROP的发生具有相关性。
Correlation was not found between ROP prevalence and oxygen inhalation mode, surface active substance, administration of dopamine and dexamethasone, and mother conditions.
目的观察地塞米松糊剂与雷公藤多甙片联合治疗口腔充血糜烂型扁平苔癣（OLP）临床疗效。
Objective To study the curative effects of Tripterygium wilfordii polyglycoside combined with dexamethasone paste for congestive oral lichen planus (OLP).
地塞米松这种人工合成的糖皮质激素，被认为在与位于垂体前叶的糖皮质激素受体结合后使ACTH分泌减少，从而最终导致皮质醇产生减少。
Dexamethasone, a synthetic glucocorticoid, is believed to bind to the glucocorticoid receptors located in the anterior pituitary, thereby reducing ACTH secretion, and ultimately cortisol production.
讨论了地塞米松促进小鼠前胃上皮增生、癌变与免疫功能低下及细胞代谢受抑制有关。
DXM would promote the carcinogenesis and the proliferate in the mouse's forestomach. The low function of the immune is related to the metabolic inhibition cf the cells.
结果超生理剂量地塞米松可抑制骨骼肌细胞的增殖，降低其SOD水平，提高MDA水平，并造成细胞的损伤与钙超载。
Results Dexamethasone beyond physical dosage could suppress cells proliferation, reduce SOD level, increase MDA, and cause cells injury and Ca 2+ overloading.
结果超生理剂量地塞米松可抑制骨骼肌细胞的增殖，降低其SOD水平，提高MDA水平，并造成细胞的损伤与钙超载。
Results Dexamethasone beyond physical dosage could suppress cells proliferation, reduce SOD level, increase MDA, and cause cells injury and Ca 2+ overloading.
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