在原发性高血压肾毁坏的病发机造中,高动力循环(hyperkinetic circulation)以及血管发作的适应性变换是基原始动要素。
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On the basis of "forward" mechanism, PHT patients manifest hyperdynamic circulation. The increased splanchnic blood flow raises portal flow, which contributes to the maintenance of elevated portal pressure.
后向血流学说认为肝内外门脉阻塞引起门脉阻力增加,导致门脉高压;前向血流学说认为门脉高压患者常表现为全身高动力循环,内脏血流增加使门脉血流相应增加,门脉高压得以持续存在。
参考来源 - 门脉高压症形成中门脉系统生物力学特性的实验研究·2,447,543篇论文数据,部分数据来源于NoteExpress
物质对维持门脉高压高动力循环起重要作用。
Substance P may contribute to the maintenance of the hyperdynamic circulation.
脾增大,脾血流量增加,脾处于高动力循环状态。
Splenomegaly and increased blood flow indicates a high level of hemodynamics in the spleen.
结论:门静脉高压症大鼠存在高动力循环状态(HCS)。
CONCLUSION: The portal hypertensive rats are in hyperdynamic circulatory state (HCS).
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