本实验提示巴曲酶对脑缺血再灌流损伤所引起的细胞凋亡有抑制作用。
The present study strongly indicate that batroxobin inhibits apoptosis induced by cerebral ischemia reperfusion injury.
目的探讨短暂脑缺血再灌流后突触传递功能的改变和神经系统的可塑性。
Objective To investigate the synaptic transmission function and neural plasticity after transient ischemia followed by repufusion.
用凝胶电泳及原位末端标记研究大鼠脑缺血再灌流后神经元的凋亡与坏死。
This article reports the apoptosis and necrosis of neurons following ischemia reperfusion in the brain of rats.
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