肾小球形态上表现为：MCN、FSGS、弥漫性系膜硬化(diffuse mesangial sclerosis，DMS)、塌陷性肾小球病变(collapsing glomerulopathy，CG)。
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目的：探讨肾疏宁防治系膜基质硬化的细胞分子机制。
Objective: To study the cellular and molecular mechanism of Shenshuning (SSN) in preventing mesangial sclerosis.
系膜节段性硬化和球囊粘连均见于重度病例；
The mesangial segmental sclerosis and capsular synechia are all happened in severe cases.
肾小球系膜细胞(GMC)的增殖通过影响细胞外基质(ecm)的分泌和对肾小球毛细血管的机械损伤而在肾小球硬化过程中发挥着重要作用。
BackgroundThe GMC proliferation plays an important role in glomerular sclerosis by influencing the ECMs secretion and mechanical injury on the glomerular capillaries.
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