AR与BA的发病机制至今仍不十分清楚，其是涉及多种炎性细胞的相互作用，诸多炎性介质、细胞因子、转录因子等共同参与的慢性炎症反应。
The pathogenesis of AR and BA has not been quite clearly, it may be involved in the interaction of many inflammatory mediators, cytokines and transcriptional factors.
牙周优势菌lps作用于单核巨噬细胞，诱导产生各种细胞因子和炎性介质，参与牙周病的炎症激活和组织创伤。
LPS involves in the inflammatory process and tissue destruction during periodontal diseases through inducing mononuclear macrophages to produce various cell factors and inflammatory mediators.
结论通心络对大鼠佐剂性关节炎具有显著抑制作用，其作用机制可能与抑制前炎症细胞因子生成、抗氧化等有关。
ConclusionTongxinluo capsule has obvious inhibition effect on AA, and the action mechanism may be related with the antioxidation and inhibition of the production of proinflammatory cytokines.
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