ROS通过氧化线粒体心磷脂、线粒体dna和线粒体重要的蛋白质对线粒体造成氧化损伤，进而诱导细胞凋亡。

Through oxidizing the mitochondrial cardiolipin, mitochondrial DNA and important proteins in mitochondria, ROS leads to the oxidative stress towards mitochondria, further induces the cell apoptosis.

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结果：磷脂酰胆碱能有效提高心、脑组织超氧化物歧化酶(SOD)活性，降低过氧化脂质(L PO)和脂褐素含量。

RESULTS: Phosphatidyl choline (PC) could increase the activity of SOD in heart and brain tissues, decreased the content of LPO and lipofuscin.

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