恶性贫血是因胃黏膜萎缩、胃液中缺乏内因子，使维生素B12吸收出现障碍而发生的巨幼细胞贫血。发病机制不清楚，与种族和遗传有关。90%左右的患者血清中有壁细胞抗体，60%的患者血清及胃液中找到内因子抗体，部分患者可出现甲状腺抗体。恶性贫血可见于甲状腺功能亢进、慢性淋巴细胞性甲状腺炎、类风湿关节炎等疾病。恶性贫血的治疗为补充维生素B12，需要终生维持治疗。
这类论文声称，每周吃超过一公斤西兰花的人晚年患恶性贫血的几率是那些吃得少的人的1.17倍。
The kind of paper that purports to show that people who eat more than one kilo of broccoli a week were 1.17 times more likely than those who eat less to suffer late in life from pernicious anaemia.
缺乏内源因素可导致恶性贫血。
恶性贫血，有巨卵形红细胞、多叶核嗜中性粒细胞。
This hypersegmented neutrophil is present along with macro-ovalocytes in a case of pernicious anemia.
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