测定休克前后及给药后心肌收缩功能和动脉血气变化。
The myocardial systolic function and arterial blood gas were observed.
离体大鼠心衰试验表明，与对照药哇巴因比较，化合物1能增加冠脉血流量和增强心肌收缩力。
Bioactivity test on isolated rat heart failure model suggested 1 could increase coronary blood flow and myocardial con - tractive power compared with positive control ouabain.
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