结论：卡托普利具有的电生理作用是由于它降低外向延迟整流钾电流及延长APD30、APD50和ERP。

CONCLUSION: Captopril exerts electrophysiologic action due to decreasing delay outward rectification potassium current and prolonging action potential duration of APD30, APD50 and ERP.

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目的：研究高温状态时下丘脑延迟整流钾通道特性的改变，探讨高温条件下出现热损伤的作用机制。

Objective:To explore the changes in properties of delayed rectifier potassium channels in hypothalamic neurons when exposed to high temperature and the mechanism of heat injury.

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目的：探讨卡托普利对豚鼠心肌细胞动作电位间期及延迟整流钾电流的作用。

AIM: to evaluate the effect of captopril on action potential duration and outward delayed rectification potassium current (Ik).

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