它们被认为是生物反应的调节者，具有有激活巨噬细胞和T细胞的能力，并产生细胞因子、白细胞介素和肿瘤坏死因子。
They are reported to serve as biological response modifiers with the capability to activate macrophages and T-cells, and to produce cytokines, interleukins and tumor necrosis factors.
牙周优势菌lps作用于单核巨噬细胞，诱导产生各种细胞因子和炎性介质，参与牙周病的炎症激活和组织创伤。
LPS involves in the inflammatory process and tissue destruction during periodontal diseases through inducing mononuclear macrophages to produce various cell factors and inflammatory mediators.
研究者还发现2型纤维蛋白溶酶原激活因子抑制物(PAI2)(一种丝氨酸蛋白酶抑制物)抑制结核杆菌感染的巨噬细胞内的膜联蛋白- 1的蛋白水解作用。
The researchers also found that plasminogen activator inhibitor type 2 (PAI2), a serine-protease inhibitor, inhibited proteolytic truncation of annexin-1 in macrophages infected with M. tuberculosis.
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