目的探讨左旋多巴治疗帕金森病诱发羟自由基增高的机制与防治的药物。

Objective To investigate the mechanism and prevention of increasing hydroxyl radical induced by L-DOPA treatment in Parkinson's disease.

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且可定向提供生成左旋或左旋芳香醇，因此可用于合成各种具芳基胺醇类结构的手性药物的重要中间体。

Furthermore, it can be oriented to produce levo-aromatic and dextro-aromatic alcohols, being an important intermediate to synthesize clural medicines of different arylamino alwhol structures.

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