目的观察氟中毒对神经细胞中尼古丁受体亚单位蛋白和基因表达水平的影响及氟中毒引起尼古丁受体功能改变的发生机制。

Objective to investigate the influence of fluorosis on nicotinic acetylcholine receptors (nAChRs) in protein and gene levels in SH-SY5Y cells and the mechanism of the receptor modification.

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免疫细胞化学方法观察了GABAA受体(gabaar)亚单位、GABA和GAD在大鼠三叉神经尾测亚核内的分布及其匹配关系。

The distribution and match relationship of CABAA receptor (GABAAR) subunits and GABA, GAD were observed in the caudal trigeminal nucleus of rat with immunocytochemistry method.

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载体经修改后包括带有一合成的剪接供体的一个异种内含子， 此供体经促性腺激素a亚单位基因（含有一个剪接受体）的内含子A中某片段的上游改造。

The vector was modified to include a heterologous intron with a synthetic splice donor engineered upstream of a fragment from intron A of the gonadotropin a subunit gene(containing a splice acceptor).

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