不过，酪氨酸激酶是直接参与还是通过前列腺素和一氧化氮合酶对糖尿病炎症产生反应的，我们目前还不知道。
However, the involvement of tyrosine kinase either directly or through the prostaglandins and nitric oxide synthase in response to inflammation during diabetes is not completely understood.
本文介绍了前列腺素内过氧化物合酶2基因的结构、表达、调控机理及其与繁殖性能的关系。
In this paper, we briefly introduced the structure, expression, regulation mechanism of prostaglandin-endoperoxide synthase 2 gene and the relationship between this gene and reproductive performance.
感染引发早产的机制与前列腺素的合成、细胞因子的异常分泌、基质金属蛋白酶活性增加等有关。
The mechanism of premature delivery caused by infection involves in increased prostaglandin synthesis, abnormal secretion of cytokines and increased activity of matrix metalloproteinases.
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