...／翟 飞文《 飞净 蕊 华南师范大学硕士论文 有关这方面的报导只见到一篇，即J．P．Sauvage的二聚轮烷分子肌（Molecular Muscle）嘲。尽管它可以通过与金属离子的络合与去络合来控制其伸缩运动，但这种 操作具有很大的局限性且不实用。
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首先，他引入了一种能够导致肌生成抑制素的受体受到抑制的基因，使肌肉细胞的细胞膜表面的分子收到停止生长的信息。
First, he has introduced a gene that turns out a stunted version of the myostatin receptor, the molecule that sits in the surface membrane of muscle cells and receives the message to stop growing.
随着时间推移，很明显RNA在强直性肌营养不良中起了核心作用，RNA是一种类似于dna普遍存在的分子。
Over time it became apparent that a central player in myotonic dystrophy was RNA, a versatile molecule that is very similar to DNA.
目的对导致肌营养不良症的基因编码产物进行检测，从分子水平为临床诊断和分型提供依据。
Objective To provide a useful tool based on molecular level for clinical diagnosis and classification by detecting gene products encoded by disease genes of muscular dystrophy.
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