AIM to investigate osteocyte apoptosis in the trabecular bone of rats with spinal cord injury (SCI) and its role in the pathogenesis of osteoporosis following SCI.
目的探讨脊髓损伤(SCI)对骨细胞凋亡的影响及其在SCI后骨质疏松发病中的可能机制。
To understand how Lrp5 influences bone properties, we generated mice with osteocyte-specific expression of inducible Lrp5 mutations that cause high and low bone mass phenotypes in humans.
为了研究Lrp5如何影响骨特性，我们建立了骨细胞特异性表达诱导型Lrp5基因突变的小鼠，该基因突变可导致人类高和低骨量表型。
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