Adenosine stimulated a significant arteriolar nitric oxide (NO) production and dilation, which were sensitive to NO synthase inhibitor L-NMMA.

腺苷能够显著激发动脉一氧化氮（NO）的产生而使血管舒张，且对一氧化氮合酶抑制剂L - NMMA 十分敏感。

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Moreover, pre-treatment of wild type mice with a nitric oxide (. NO) synthase inhibitor prevented the HBO2-induced elevation in stem cell factor and circulating stem cells.

而且，野生型小鼠用NO合酶抑制剂预处理后阻止了HBO2介导的干细胞因子和外周血干细胞的增加。

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