The story starts at the molecular level within tiny nerves that conduct pain signals.

而这一切从痛觉神经的分子水平开始。

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Conclusion. The genes identified in this study, especially those involved in pain signaling and inflammation, serve as potential targets for molecular-based therapy for lumbar radiculopathy.

结论：本研究中鉴别的基因尤其是疼痛信号和炎症相关基因可能是腰神经病分子治疗的潜在靶点。

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These results strongly suggest that chronic pains may have molecular and cellular mechanisms which are different from nociceptive pain and acute pain.

这些结果有力地提示，慢性痛具有有别于伤害性痛和急性痛的分子和细胞机制。

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