临床麻醉学杂志投稿系统 关键词: 七氟醚后处理;海马脑片;缺氧无糖损伤 [gap=1011]Keywords: Sevoflurane postconditioning; Hippocampal slices; Oxygen-glucose deprivation injury
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中枢效应机制:1994年,Melchers等[8]用大鼠脑海马薄片(hippocampal slices)离体标本,进行了HI-6地脑内非AChE重活化效应机制地研究。
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Conclusion: (1) Seizure-like activity could induce neurons apoptosis in cultured hippocampal slices.
结论:(1)痫性发作可以增加体外培养海马脑片神经细胞凋亡发生,加重神经元损伤。
参考来源 - 脑源性神经营养因子对培养大鼠海马脑片痫性发作的作用及机制研究·2,447,543篇论文数据,部分数据来源于NoteExpress
In acutely isolated hippocampal slices, kainate application increased S100B secretion in a neural-activity-dependent manner.
在急性海马的脑片中,给予红藻氨酸盐可以以神经元依赖的方式促进S100 B分泌。
Conclusion Potassium channel blocker IBTX can alleviate the injury of neurons in ischemic hippocampal slices by decreasing the apoptosis of neurons.
结论钾通道拮抗剂IBTX可以减轻低氧海马脑片的损伤,其作用机制可能与减少细胞凋亡有关。
Methods: By way of the electrophysiological recording technique, we observed the influence of L-NNA on orthodromic population spike(OPS) during anoxia in rat's hippocampal slices.
方法:在大鼠离体海马脑片,用电生理记录技术,观察L-NNA对缺氧时海马脑片顺向群峰电位( OPS)的影响。
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