Objective To ascertain related bioactivity mediums of silica-reduced pulmonary fibrosis in rats, to construct the regulator network of the material bioactivity and its genes as well as both integration, to evaluate network regulatory mechanism of silicosis fibrosis systematically.
目的确定矽肺纤维化相关的生物活性介质,构建生物活性介质调控网络及其基因调控网络,以及二者整合网络,以此系统阐明矽肺纤维化的网络调控机制。
参考来源 - 矽肺纤维化生物活性介质调控网络研究The article is written to investigate the storage model of silicosis fibrosis bioactive medium, via give a research in NXD theory, and via analyze the main NXD.
本文旨在通过对NXD理论的研究,和对主流NXD的分析,来探讨矽肺纤维化生物活性介质数据的存储模型。
参考来源 - 基于NXD的矽肺纤维化生物活性介质数据库的构建与研究·2,447,543篇论文数据，部分数据来源于NoteExpress
Objective To investigate long term therapeutic effect of combined anti fibrosis drugs on silicosis.
目的研究矽肺抗纤维化治疗的远期疗效。
The basic pathological change of silicosis is the lung interstitial fibrosis and the formation of silicotic nodule.
矽肺最基本的病理变化是肺间质纤维化和矽结节的形成。
Similar to the silicosis, idiopathic pulmonary fibrosis (IPF), which is another lung interstitial disease, is characterized by lung fibrosis.
与矽肺的发病相似，特发性肺纤维化(IPF)也是以肺成纤维细胞大量增殖为主要特征的肺纤维化疾病。
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