Contrast the pathological change and biochemical indicator detection condition in rat lung tissue circa medicine intervention, decision the curative effect.
对比药物干预前后大鼠肺组织的病理变化情况及生化指标的检测情况，判断疗效。
Conclusions Aging could accelerate fibrin deposition in pulmonary tissue of LPS-induced rat model of acute lung injury, and promote the injury of endothelial cell function caused by fibrin deposition.
结论增龄能够促进脂多糖诱导的急性肺损伤大鼠肺组织纤维蛋白沉积，增强纤维蛋白的内皮细胞损伤作用。
AIM: to investigate the expression of interleukin-13 (IL-13) in lung tissue of rat asthmatic model and effect of triptolide on expression of IL-13.
目的：探讨白细胞介素13 (IL - 13)在大鼠哮喘模型肺组织中的表达以及雷公藤内酯醇对IL - 13表达的影响。
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