Conclusion the shapes of the excitatory frequency response areas of the neuron in the primary auditory cortex are various and might result from the same mechanisms.

结论大鼠初级听皮层神经元的兴奋性频率反应域具有多种类型，而不同的兴奋性频率反应域的形成机制可能相同。

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Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.

结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。

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The pathophysiological mechanisms of ischemic cerebral injury involved in the release of excitatory amino acids, depolarization about the infarction, inflammatory reaction and neuron apoptosis.

缺血性脑损伤的病理生理机制包括兴奋性氨基酸的释放、梗死灶周围去极化、炎症反应及神经元凋亡等。

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Now, these signals can be either excitatory, which is that they raise the likelihood the neuron will fire, or inhibitory in that they lower the likelihood that the neuron will fire.

这些刺激信号可能是兴奋性的,会增加神经元产生神经冲动可能性,也可能是抑制性的,会降低神经元产生神经冲动的可能性

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