The inhibitor for NO or for MMPs significantly reduced the LSEC IRI, suggesting that NO and MMPs are important mediators in the LSEC IRI.
应用一氧化氮和基质蛋白酶的抑制剂明显降低肝窦内皮细胞的损伤，表明一氧化氮和基质蛋白酶在缺血-再灌注损伤中是重要的介导物。
The tissue inhibitor of metalloproteinases (TIMPs) are a large family of multi-functional genes, and they specifically inhibit MMPs.
基质金属蛋白酶的组织特异性抑制物是一组多功能基因家族，它们能特异性地抑制基质金属蛋白酶。
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