Increased EGR1 expression activates transcription of other signaling molecules, including CDK5 and tyrosine hydroxylase, and exerts long term effects on neural cell growth and differentiation (2, 3).
增加EGR1表达可以激活其他信号分子的转录，包括CDK5和酪氨酸羟化酶，对神经细胞生长和分化有长期影响(2,3)。
In experiments on mice, researchers tracked brain levels of cell-signaling protein molecules called cytokines, which are boosted by SSRIs, and found they were counteracted by anti-inflammatory drugs.
研究人员在白鼠上实验，追踪叫做细胞活素类的细胞信号传导蛋白的脑部水平(它受ssris的刺激)，发现消炎药削弱了这些蛋白质的活动。
Next we tried to find out which signaling molecules in the selected three tumor cell lines are related to their resistance to anoikis.
对于筛选出的三种抗脱落凋亡的肿瘤细胞系，它们的抗脱落凋亡特性究竟与哪个或哪些信号分子有关呢？
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