晚期糖基化终产物(advanced glycation end products,AGEs)是一组在蛋白质的氨基和糖的醛基之间非酶性糖基化反应终产物的总称[2].
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晚期糖基化终末产物(advanced glycation end products,AGE)是指蛋白质、脂质或核酸等大分子在没有酶参与的条件下,自发地与葡萄糖或其他还原单糖发生非酶糖基化反应,...
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背景 晚期糖基化终产物(Advanced glycation end products，AGEs)是指蛋白质、核酸或脂质等与大分子物质的氨基在不需酶参与条件下，能自发地与葡萄糖或其它还原糖的醛基或酮...
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晚期糖基化终末产物(advanced glycation end products, AGEs)是蛋白质的氨基酸、还原糖的醛基之间发生非酶促糖基化反应的终产物,AGEs的形成过程是糖化和氧化的共同过程；由于肾脏结构和...
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Advanced glycation end products AGEs 糖基化终末产物 ; 糖基化终产物
advanced glycation end products AGE 化终产物
receptor for advanced glycation end-products 高度糖基化终产物受体 ; 糖基化终产物受体
AGEs advanced glycation end products 晚期糖基化终末产物
advanced glycation end-products age 晚期糖基化终产物
AGEs -advanced glycation end-products 糖基化终产物
advanced glycation end products receptors 晚期糖基化终产物受体
advanced glycation end products inhibitor 糖基化终末产物抑制剂
Four key factors were implicated in diabetic complications: increased polyol pathway （hyperglycemia-induced activity aldose reductase）, increased formation of advanced glycation end products （AGE）, hyperglycemia-induced activation of protein kinase C （PKC） isoforms and oxidative stress.
糖尿病并发症的主要发病机制有醛糖还原酶活性增强、糖基化终末产物（AGE）形成增多、PKC活性增加以及氧化应激。
参考来源 - 硝克柳胺防治糖尿病并发症作用机制研究·2,447,543篇论文数据，部分数据来源于NoteExpress
An elevated level of skin advanced glycation end products (AGE) is a biomarker of diabetes, the authors explain.
作者解释道，升高的皮肤晚期糖基化终末产物(age)是糖尿病的生物标志物。
The excess in blood glucose reacts with proteins in tissues to form what are known as advanced glycation end products, or AGE's.
过量血糖反应蛋白在组织中，以何种形式被称为晚期糖化终产物，或年龄的。
Objective: To study the effects and the mechanism of advanced glycation end products on bovine vascular smooth muscle cell in vitro.
目的：探讨晚期糖化终产物对离体新生牛胸主动脉血管平滑肌细胞的影响及机制。
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