The mechanism of the acute tubular necrosis (ATN) caused by acute renal ischemia-reperfusion injury is complicated and there are many correlation factors in it.

急性缺血再灌注引起急性肾小管坏死发生机制较为复杂，相关因素比较多。

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This is an acute renal infarction. Note the wedge shape of this zone of coagulative necrosis resulting from loss of blood supply with resultant tissue ischemia that produces the pale infarct.

急性肾梗死，属凝固性坏死，坏死区域呈楔形，是由于组织血供减少引起缺血，终而产生灰白的梗死区。

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Animal ARF can be induced by renal ischemia-reperfusion injury, glycerine, gentamicin and mercuric chloride, and typical acute tubular necrosis can be observed in pathological changes.

肾动脉钳闭引起的肾脏缺血再灌注以及甘油、庆大霉素、氯化汞均可致实验动物急性肾衰竭，病理学上可见典型的急性肾小管坏死。

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