结论铁过负荷增加脂质过氧化并易化细胞凋亡，加重免疫性肝损伤。
Conclusion the immunological liver injury could be aggravated by iron overload through catalyzing lipid peroxidation and facilitating the apoptotic process of hepatocyte.
结论：尼莫地平可能通过抑制HO - 1表达、维持铁离子稳态，减轻铝过负荷所致的神经元退行性变。
CONCLUSION: Nimodipine relieves the neurodegeneration induced by aluminum overload through inhibiting the expression of HO-1 and keeping the homeostasis of iron.
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