诱导型一氧化氮合酶的诱导表达往往导致心肌的收缩功能下降。
The up regulation of inducible nitric oxide synthase ( iN OS ) often negatively modulates myocardial function.
诱导型一氧化氮合酶可以作为缺氧程度和肺组织损伤程度的标志之一。
Inducible nitric oxide synthase may be one of the signs at degree of hypoxia and damnification of lung tissue.
这一结果为诱导型龙胆酸酶的进一步研究打下了基础。
The results obtained provide us clues for further investigation of inducible GDO enzyme.
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