这使得确定的重要细节在其复制周期和病毒如何与宿主相互作用。
This has allowed identification of important details in its replication cycle and how the virus interacts with its host.
结论HCMV的感染改变了细胞内P 53的水平，可能影响其在细胞周期中的正常功能，营造有利于病毒复制的环境。
Conclusion HCMV induces elevated levels of P53, which may affect its normal function in regulating checkpoints of cell cycle, and facilitate its own replication in host cell.
细小病毒的复制紧紧依赖于和增殖及分化有关的细胞因子，尤其是和细胞周期S-期有关的因子。
The replication of parvovirus is strongly dependent on cellular factors associated with proliferation and differentiation, especially those related to the S-phase of the cell cycle.
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