海马神经元损伤是神经突触可塑性调节中重要的一个环节，在抑郁症发病机制的研究中占有重要地位。
The damage of hippocampus neuron is the key part in plasticity regulation of synapse and play a critical role in the mechanism of depression.
通过对出生前有慢性应激经历子代海马CA 1区LTP和LTD诱导性及学习记忆和行为反应的测定，探讨出生前慢性应激对子代海马CA 1区突触可塑性及学习记忆行为的影响。
We explored the effects of prenatal stress on hippocampal synaptic plasticity and behavior of the offspring by testing LTP and LTD in hippocampal CA1 area and learning and memory in Morris Water Maze.
已有的电生理学实验已经表明，褪黑素可以调节海马神经元的兴奋性和突触可塑性。
Previous electrophysiological experiments have revealed that melatonin modulates hippocampal neuronal excitability and plasticity.
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