可以用非去极化的神经肌肉阻滞剂(如维可罗宁、卡肌宁或潘可罗宁)代替琥珀胆堿，但麻醉的出现将延迟60秒钟。
A non - depolarizing neuromuscular blocking drug (e. g., vecuronium, atracurium, or pancuronium) can be substituted for succinylcholine, but the onset of paralysis is delayed by about 60 seconds.
结果提示，运动试验诱发VT的机制，可能是延迟后去极化触发活动和肾上腺素敏感性增高自律性异常。
Combining with electrophysiological results, we suggest that the mechanism of inducing VT might he due to triggered activity and abnormal automaticity.
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