...增强心脏的电稳定性 ］和陈魁等［2］的实验表明，缺血预处理(ischemic preconditioning, IPC)对缺血再灌流(ischemic reperfusion, I/R)心肌的保护作用呈两个不连续的时相：早发的保护作用在2～3 h后消失，迟发的保护作用在24 h左右又可...
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本实验提示巴曲酶对脑缺血再灌流损伤所引起的细胞凋亡有抑制作用。
The present study strongly indicate that batroxobin inhibits apoptosis induced by cerebral ischemia reperfusion injury.
结论再灌流前应用山茛菪碱能减少再灌流后氧自由基的产生，可减轻缺血—再灌流对移植肝脏的损伤。
Conclusions the use of anisodamine before reperfusion could reduce the production of oxygen derived free radical, therefore alleviate the injury of grafted liver caused by ischemia reperfusion.
环氧化酶- 2 (COX - 2)及其代谢产物可通过对脉管系统、血脑屏障和神经元本身的损伤，参与脑缺血再灌流后损伤的机制。
Cyclooxygenase-2 (COX-2) and its metabolic products can be involved in the mechanism of the ischemic brain injury through destructing the vascular system, blood-brain barrier and the neuron.
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