...在，但对病原体有较强抗性的植物在感染后的几小时内活性氧持续增加，并出现超敏反应(HR)和细胞程序性死亡(programmed cell death, PCD)，从而抑制病原体的进一步侵染。
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近年来，在PD CD 4基因对程序性细胞死亡的影响，以及对转化和蛋白质翻译的抑制作用的相互关系等方面进行了大量研究。
Quantity of studies of PDCD4 have been carried out about its effect on programmed cell death and correlationship with inhibition for transformation and protein translation in recently year.
在病原、诱发因子和激素应答中是调节细胞程序性死亡的关键因子。
It is the key factor regulating programmed cell death in pathogen, evocator and hormone responses.
应用DNA凝胶电冰、电镜观察和流式细胞仪分析，对加热诱导G422胶质瘤程序性细胞死亡的作用进行了研究。
Using DNA gel electrophoresis, electron microscopic examination and flow cytometric analysis, program cell death induced by hyperthermia in mouse G422 glioma in vivo was studied.
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