为了进一步支持我们的假设，我们还采用大鼠内毒素耐受模型进行了研究。
In order to further support our hypothesis, endotoxin tolerance model has been used in our studies.
结论HIC呈内毒素无反应性，其表面内毒素相关受体tlr4、CD 14和MD 2不表达是其耐受内毒素作用的重要分子机制。
Conclusion There was no response of HICs to LPS stimulation, and no expression of TLR4, CD14 and MD-2 on HICs may be an important molecular mechanism by which they tolerated to lipopolysaccharide.
应用推荐