塞可西丁可促进蛋白质合成和增强消化酶的活性；
Secxitin can promote the synthesis of protein and enhance the activity of digestive enzyme.
实验结果表明，DEG的抗溃疡作用可能与增加胃内PGE_2含量、抑制胃液分泌和促进蛋白质合成有关。
To sum up, the prineiple mechanism of anti-gastric ulcer of DEG may be by raising the levels of PGE2 in gastric juice and inhibiting pepsinia and promoting synthesis of protein.
炎性细胞因子，如白细胞介素1和白细胞介素- 6对合成急性期蛋白质(6,7)有促进作用。
Proinflammatory cytokines such as interleukin-1 and interleukin-6 have stimulatory effects on the synthesis of acute phase proteins (6, 7).
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