To examine this pathway in more detail, the researchers deleted the DDAH gene in mice.

为了更具体的验证这个途径，研究者把小鼠的DDAH基因敲除。

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They also designed specific inhibitors (small molecules) which bind to the active site of human DDAH.

他们也设计了一个特殊的抑制物(小分子)，它可以结合在人类DDAH的活性位点上。

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Genes and their pathways are crucial to our understanding of cardiovascular disease and a better understanding of DDAH-1 could lead to important new treatments.

探索这些基因和它们的途径，对我们了解心血管疾病和更好地了解DDAH - 1可导致新疗法的产生很重要。

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