The results indicated the upward regulation of TR in rat brain during acute cerebral ischemia is a compensatory response, which allows the maximum binding of thyroid hormones.
大鼠脑缺血急性期脑甲状腺激素受体向上调节为机体的重要代偿反应。
These alterations of nestin positive cells persisted up to 6 weeks post-ischemia, and then, the nestin positive response in the ischemic brain decreased gradually.
皮质缺血区和尾壳核区的巢蛋白阳性反应持续到脑缺血6周。随后，其反应稍有减弱。
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