结论AT P不仅抑制人胃癌细胞的增殖,而且诱导分化,使人胃癌细胞的恶性表型向正常表型逆转。
Conclusion ATP not only inhibits proliferation but also induces differentiation of a human gastric cancer cell line with reversion from malignant toward normal phenotype.
我们假设这样一种复杂的表型逆转可能是由于多重信号因子(彼此结合时可允许肿瘤细胞形成静态类腺泡结构)的作用。
We hypothesized that such a complex phenotypical reversion is likely the result of multiple signaling factors that in combination allow cancer cells to form quiescent acinar-like structures.
对HSC起源、分布与表型转变的研究将为治疗与逆转肝纤维化提供新的方向。
The study of HSC on the origin, distribution and phenotypic changes will provide a new direction for the treatment and reversal of hepatic fibrosis.
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