一氧化氮作为一种气体信使分子，参与神经系统的信息传递，对海马、小脑等神经元突触的可塑性和神经网络的构建产生重要影响。
As a gas message molecule, nitric oxide takes part in the information transfer in nervous system, which affects plasticity of synapse in hippocampus, and cerebellum and construction of neural network.
目的探讨短暂脑缺血再灌流后突触传递功能的改变和神经系统的可塑性。
Objective To investigate the synaptic transmission function and neural plasticity after transient ischemia followed by repufusion.
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