结论PI 3 K信号通路可能通过促进气道平滑肌细胞增殖参与哮喘气道高反应性及气道重构过程。
Conclusion the PI3K signal pathway may be involved in AHR and the airway remodeling through promoting airway smooth muscle cells proliferation in asthma.
结论：缬沙坦可能是通过抑制AT 1受体抑制哮喘气道炎症和降低气道反应性。
Conclusion: Valsartan may inhibit airway inflammation and reduce airway responsiveness by suppressing AT1 receptors.
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