类风湿病中左心室收缩功能障碍：一种未被认识的负荷？
Left ventricular systolic dysfunction in rheumatoid disease: An unrecognized burden?
结论：毛花甙C的强心作用与迷走神经兴奋作用可能分别参与了阵发性房颤伴有与不伴有左心室收缩功能障碍患者的复律机制；
Conclusion:The cardiotonic and parasympathetic stimulating effects of deslanoside probably take part in the defibrillation of PAf with and without SI respectively.
因此，我们检测了左心室收缩功能障碍并继发肿瘤坏死因子(TNF1.6)过表达的转基因老鼠腺苷水平和选择性ar的表达。
Therefore, we assessed adenosine levels and selective ar expression in transgenic mice with left ventricular systolic dysfunction secondary to overexpression of tumor necrosis factor - (TNF 1.6).
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