同样，在卵巢癌小鼠模型中，抑制应激和相关的去甲肾上腺素和肾上腺素增加可防止肿瘤细胞发生失巢凋亡，从而促进肿瘤生长。
Similarly, in a mouse model of ovarian cancer, restraint stress and the associated increases in norepinephrine and epinephrine protected the tumor cells from anoikis and promoted their growth.
密歇根州大学实验室开发的这一新的小鼠模型是基于人卵巢肿瘤细胞分子缺陷的特点，本研究第一作者Kathleen R。
The new mouse model developed in the U-M lab is based on molecular defects shown to be present in human ovarian tumor cells, says senior author Kathleen R.
他们发现这些细胞扮演着一种基本细胞的角色，首先从小鼠的卵巢肿瘤分离得到，而后又在人的癌细胞中鉴定出类似的细胞。
They found these stem cells, which act as a kind of master cell, first in batches of ovarian tumor cells taken from mice. They then identified similar cells in human cancer cells.
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