产生LTP的生物学基础之一是海马神经元突触上的N -甲基- D -天门冬氨酸(NMDA)受体。

And the N-methyl-D-aspartate (NMDA) receptor on the synaptosome in hippocampus is one of the biological bases of LTP.

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目的：利用N -甲基- D -天门冬氨酸(NMDA)损伤小脑颗粒神经元的模型，探讨IL - 6对神经损伤的保护作用，为临床上应用细胞因子防治脑损伤提供新思路。

Objective: by using a cellular model to acute damage to cultured cerebellar granule neurons induced by N-methyl-D-aspartate (NMDA) to explore neuroprotective effect of IL-6.

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