它还可以降低高半胱氨酸中的血压。
高半胱氨酸是一种氨基酸。
高浓度的高半胱氨酸会导致患老年痴呆症的风险增加。
High levels of homocysteine have been linked to a greater risk of Alzheimer's.
高半胱氨酸中的血压过高可以导致心脏病和中风。
High homocysteine levels have been linked to heart attacks and strokes.
维生素b可以帮助阻止高半胱氨酸,它是损害血管的一种氨基酸。
B vitamins can help stifle homocysteine, an amino acid that damages blood vessels.
实验开始时高半胱氨酸含量最高的受益最大,脑萎缩减少了50%。
Those who started the trial with the highest levels of homocysteine experienced the greatest benefit - 50% less brain shrinkage.
选用维生素是因为他们控制了血液中一种叫高半胱氨酸的氨基酸的含量。
The vitamins were chosen because they control the amounts of an amino acid called homocysteine in the blood.
口服叶酸和维生素b12可以降低血浆中总高半胱氨酸的水平。
Oral administration of folic acid and vitamin B12 can lower plasma total homocysteine levels.
背景——升高的总高半胱氨酸水平与较高的静脉血栓栓塞形成风险相关。
Background: Elevated total homocysteine levels are associated with a higher risk for venous thromboembolism.
在过去15年期间对高半胱氨酸的研究和涉及它的新陈代谢的维生素已经变得非常动态。
During the last 15 years the research on homocysteine and the vitamins involved in its metabolism has become very dynamic.
TMG通过中和有害的高半胱氨酸来保护心血管系统,并提供有价值的营养支持健康的肝功能。
Trimethylglycine (TMG) protects the cardiovascular system by neutralizing harmful homocysteine and provides valuable nutritional support for healthy liver function.
结论——通过叶酸,维生素b6和维生素b 12降低高半胱氨酸水平,并没有降低症状性静脉血栓形成的风险。
Conclusion: Decreasing homocysteine levels with folic acid and vitamins B6 and B12 did not reduce the risk for symptomatic venous thromboembolism.
综述近年来国内外有关S- 腺苷高半胱氨酸水解酶分子水平的催化机制及其抑制剂的作用机制研究和产品开发状况。
The researches of catalytic mechanism of S-adenosylhomocysteine (AdoHcy) hydrolase and acting mechanism of its inhibitors and the related product developing status in recent years were reviewed.
综述近年来国内外有关S- 腺苷高半胱氨酸水解酶分子水平的催化机制及其抑制剂的作用机制研究和产品开发状况。
The researches of catalytic mechanism of S-adenosylhomocysteine (AdoHcy) hydrolase and acting mechanism of its inhibitors and the related product developing status in recent years were reviewed.
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