醛缩酶氨基酸序列保守性较高。
The results indicate that aldolase owns high conservation in its amino acid sequence.
中剂量和低剂量可显著地降低血清乳酸脱氢酶、醛缩酶和肌酸激酶活力。
The median and low doses decreased the activity of LDH, ALD, CK.
动物实验证明，尿嘧啶和尿嘧啶核苷对实验性肌强直症小鼠血清醛缩酶有降低作用。
Animal experiments demonstrated that uracil and uridine were capabie of lowering the elevated serum aldolase level of mice suffering from experimental myotonia.
在上述观察时间内，雄虫的醛缩酶、6 -磷酸甘露糖异构酶和6 -磷酸葡萄糖脱氢酶活性受一定的影响。
In male worms, only aldolase, mannose 6 phosphate isomerase and glucose 6 phosphate dehydrogenase were slightly inhibited.
醛缩酶和TNBS之间的反应支持酶分子的氨基，尤其是蛇肌醛缩酶的氨基在维护其天然构象中起着特殊作用的观点。
The reactions between aldolase and TNBS suggest that the amino group or groups, in snake aldolase, may play a special role in maintaining its native conformation.
结论肝移植术后血清醛缩酶、亮氨酸转肽酶和谷氨酸脱氢酶可作为新植入肝脏肝细胞损伤的有效指标，为临床治疗提供帮助。
Conclusion Serum ALD, LAP and GLDH levels are effective markers of hepatocellular damage after liver-transplantation and conducive to clinical therapy.
结论肝移植术后血清醛缩酶、亮氨酸转肽酶和谷氨酸脱氢酶可作为新植入肝脏肝细胞损伤的有效指标，为临床治疗提供帮助。
Conclusion Serum ALD, LAP and GLDH levels are effective markers of hepatocellular damage after liver-transplantation and conducive to clinical therapy.
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