目的研究血管活性肽在血管再狭窄形成中的作用。
Objective To investigate the effects of several vasoactive peptides on the development of restenosis.
路移植管搭桥术后，常会产生血管再狭窄，导致手术失败。
Restenosis always occurs in coronary artery bypass graft and results in the surgery failure.
目的：探讨在血管再狭窄形成过程中内膜增生及血管重塑的作用。
AIM: To explore the role of intimal hyperplasia and vascular remodeling in the formation of vascular restenosis.
当血管损伤后，局部产生的细胞因子引起炎症反应是血管再狭窄的主要原因。
The vascular restenosis is attributable to a local inflammation after vascular injury, which results in the production of cytokines.
研究表明ROCK与血管再狭窄、心肌损伤、心肌细胞凋亡及心力衰竭等有关联。
It has been shown that ROCK is associated with restenosis, myocardial damage, myocardial apoptosis, and heart failure.
目的：探讨在不锈钢冠状动脉支架上携带质粒基因，为心血管再狭窄基因治疗的临床应用提供试验依据。
Objective: to investigate stainless steel coronary stent-based plasmid gene delivery system and evaluate its feasibility and effectiveness to gene therapy of cardiovascular restenosis.
谢潮鑫，孟猛，曾伟杰，钟磊，魏春霞，罗佼心血管支架材料生物力学及生物相容性与支架置入后血管再狭窄。
Xie C. -X. , Meng M. , Zeng W. -J. , Zhong L. , Wei C. -X. , Luo J. Biomechanics and biocompatibility of cardiovascular stent materials and restenosis following stent implantation.
方法总计67只新西兰兔接受了经股动脉途径行球囊损伤腹主动脉术，术后4周处死兔，观察血管再狭窄模型制作情况。
Methods 67 New Zealand rabbits injured by dilated balloon underwent abdominal aortic operation. They were killed after 4th weeks and observed vascular stenosis model.
结论：局部转染vegf165基因可抑制血管新生内膜增生及血管再狭窄，为将来血管内膜增生的基因治疗奠定基础。
CONCLUSION: Local transfection of VEGF165 gene restrains intimal hyperplasia and restenosis of vessels, which lays a foundation for future gene therapy of vascular intimal hyperplasia.
而平滑肌细胞凋亡与动脉粥样硬化和血管成形术后再狭窄密切相关。
The apoptosis of smooth muscle cells was related to the atherosclerosis and restenosis of vessels.
血管成形术后发生再狭窄影响手术的远期疗效。
Restenosis after angioplasty influences the long dated surgical effect.
目的计算放射性球囊治疗冠状动脉再狭窄时导管内核素对正常血管的剂量分布。
Objective To estimate dose distribution in normal vessels delivered by radioactive solution in the catheter for intracoronary intra-balloon brachytherapy.
目的：观察辐射对血管活性物质的影响及其机制、意义以探讨辐射防治再狭窄的机制。
Objective: To study the effects and mechanism of radiation on various kinds of vasoactive substances.
目的：研究化痰益气活血治法对血管狭窄模型兔再狭窄相关基因表达的影响。
Objective:Influence of dissipate phlegm, invigorate vital energy, promoting blood flow treatment to restenosis related gene of rabbit angiostegnosis model was studied.
血管介入术后的再狭窄严重影响术后远期疗效。
Restenosis after vascular intervention influences the long term curative effect seriously.
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是血管成形术后再狭窄的重要原因之一。
The major complication of vascular recanalization is intimal hyperplasia which in duc mainly to over-proliferation and migration of smooth muscle cells (SMC).
目的：评估质粒介导下蛋白涂层金属支架局部转染血管内皮生长因子(VEGF)基因对小型猪冠状动脉再狭窄的作用。
Objective: to assess the effects of local vascular endothelial growth factor (VEGF) transfer using protein coated metallic stents on restenosis of coronary arteries in mini swine model.
目的评价兔髂动脉内近距离治疗预防经皮经腔血管成形术（PTA）后再狭窄的可行性及作用。
Objective To evaluate the role of intra arterial brachytherapy to prevent the iliac artery restenosis in the rabbit following percutaneous transluminal angioplasty (PTA).
本文综述了声动力疗法在肿瘤治疗、血管成形术后再狭窄等增殖性疾病方面的医学应用及相应的生物学机制。
This paper reviews the medical application of sonodynamic therapy in the treatment of tumor and restenosis after percutaneous transcoronary angioplasty and the corresponding biomechanism.
因此，降低血管受损局部基质金属蛋白酶表达将成为防治血管内再狭窄的一个方向。
Therefore, reduction of MMPs expression in the local area after vascular injury provides a direction for preventing and curing vascular restenosis.
血管内放射导致细胞死亡的主要形式为凋亡，电离辐射预防再狭窄的主要机制是平滑肌细脑的增殖抑制。
Apoptosis is a major style of cell death induced by endovascular radiation. The inhibition of SMCs proliferation induced by radiation is a main mechanism for prevention of restenosis.
这些药物涂层用于防止再狭窄过程，即动脉血管产生新阻塞。
The coatings are meant to combat the tendency of arteries to form new blockages, a process called restenosis.
P 27蛋白的高表达可显著抑制血管平滑肌细胞增殖，有望成为治疗PT CA后再狭窄的新手段。
P27Kipl restrains the proliferation of VSMC obviously and is prospective to become a new means to cure restenosis after PTCA.
防治静脉移植物再狭窄的方案包括药物治疗、基因治疗和血管外支架。
Therapeutic strategies to prevent vein graft disease include pharmacotherapy, gene therapy and external stenting.
目的观察中药对冠心病支架术后缺血心肌血管新生以及支架再狭窄的临床疗效。
ObjectiveTo observe the effect of Tongxinfang on coronary collateral formation and stent-restenosis in patients with coronary artery disease after stenting.
结论颈动脉内膜切除术后超声和血管造影联合应用可动态监测早期再狭窄过程，对临床评估有重要意义。
Conclusions Ultrasound combined with angiography are suitable for monitoring the procedure of restenosis after CEA and important for clinical assessment.
同时，烟碱还有促进动脉粥样硬化和促进血管内膜增殖的作用，因此有促进再狭窄的作用。
Nicotine can also accelerate atherosclerosis and intimal proliferation, so may contribute to the development of restenosis.
PTCA后白细胞的激活可能触发一系列病理生理联级反应，释放某些炎症介质，促进血管平滑肌增殖导致再狭窄；
Activation of leukocytes after PTCA can trigger a serial cascade of pathophysiologic reaction, releasing some inflammation intermediates lead to restenosis.
而置入支架的血管可因一种叫做再狭窄的过程而再次闭塞，这就意味着要二次手术。
The artery in which the stent is placed, however, can become blocked again during a process called restenosis. This re-closure means a repeat surgery.
术后靶血管管腔再狭窄是冠状动脉介入治疗术的主要并发症，如何有效预防再狭窄已成为目前介入心脏医学界的主要课题之一。
The in-stent restenosis is one of the main complications in coronary artherosclerotic heart disease patients after PTCA and PCI.
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